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Back Acne - Causes, Prevention & Treatment For Back Acne

Many people suffer from acne in a single form or another. Along with cosmetic acne, back acne is also quite common. It can become very embarrassing or even painful, but there are solutions: If you have been looking for a remedy for adult acne you might be willing to go in here: adult acne treatmentIf you want the best back acne please consider going to the link here: acne cystIf you want to learn how to get rid of acne go to this link : acneq.comIf you wish the best cystic acne please consider going to the link here: Navigate To This WebsiteIf you are looking for best acne treatment products visit the website at the link here: seeclogged poresIf your think a serum for acne treatment may be a sound solution go to the website here: I thought about this

Back Acne is the manifestation of injuries inflicted to cells lining the sebum canals within the hair follicles because they cannot sustain a subito outflow of sebum to the surface of the skin, or because of hardened sebum. Strong sebum outflow is caused by an increase of androgen and testosterone (2 important hormones). The hormone androgen begins production when puberty occurs and is responsible for the levels of sebum existence.

Since much sebum is produced it then begins to function its way to the surface of the skin. As the sebum travels to the hair shaft it mixes with dead cells that causes the hair follicle to get blocked. In blocked pores bacteria that reside in symbiosis with us inside the sebum glands now in an environment without oxygen start proliferating to much or uncontrolled and also produce chemicals which damage our tissue.

Studies have found links that suggest that trans-fats may break-down the body's supply of essential fatty acids. This is why acne breakouts are more likely to appear in people that have a deficiency in Omega-3 and Omega-6 oils.

The appearance of acne can be due to sebum being produced with oleic acids which can cause irritation to the cells lining the sebum ducts. It has also been suggested that oleic acid sebum is drier, firmer, hardened, and thus more prone to create lesions to the cells lining the walls of the sebum ducts and to form follicular plugs which lead to acne blemishes.

Within the follicular channel of humans you can find bacteria which are indigenous towards the follicular lining. Among the bacteria flora present are usually anaerobic, gram positive organisms called Proprionibacterium acnes. It is interesting to note that they are present in plethora in pathologically afflicted sites. They are reduced during oral antimicrobial treatment and their lack from nonhuman pet skin is stunning especially since animals do not exhibit acne vulgaris.

Proprionibacterium acnes bacteria lives around the keratin lined follicular channel in symbiosis around, thus providing us certain benefits.

Many bacteria live on our skin and mucous membranes (mouth area, genitals, sebaceous glands, etc) and supply different benefits to us. The particular benefit that resident Proprionibacterium acnes bacteria living around the keratin lined follicular canal bring to us is that they help the sebaceous glands produce the actual oil that lubricates the outer skin and keeps the actual pH of skin at a level that does not allow for other bacteria to call home on our pores and skin surface and inside the follicles.

Proprionibacterium acnes ingest the sebum made out of the sebocytes of the sebaceous glands. This nascent sebum is largely lipid in composition and also contains DNA, RNA, proteins and other cellular components that result from the breakdown associated with sebocytes themselves. The Proprionibacteriumacnes that have an affinity for lipids, feed on the particular nascent sebum.

It has been proven that Proprionibacterium acnes are only only in sweat rich areas. If the nutrients increase as a result of an active as well as large sebaceous system, then colonization as well as high growth prices of Proprionibacterium acnes will form. Then the homeowner bacterial flora can produce biologically active molecules such as histamine, extracellular enzymes and also peptides which have the effect of the movement of cell developed by our immune system in reaction individuals chemical stimulus (chemotaxis) resulting in the inflammatory infiltrate characteristic of acne vulgaris.

Proprionibacterium acnes can easily produce proteinases, lipase and hyaluronatelyase which may serve since the catalysts or initiators with the inflammatory infiltrate which includes been shown to be composed of neutrophils and also lymphocytes.

Acne starts with all the formation of a characteristic lesion: the comedo. This produces a blockage in the pilosebaceous canal following dyskeratinization with the infundibular zone from the canal. A major effect from the blockage is to change the rheology from the sebum and the actual physicochemical properties from the area. Such modification allows resident cutaneous strains to hyperproliferate which in turn triggers an inflammation reaction inside the organism.

The comedo is done by retention of layers of dead skin referred to as keratin in the lining of the hair follicles. In addition to hyperkeratosis (which is thickening or retentative layering associated with keratin), there is an accumulation of sebum which can be the lipid-laden product of the sebaceous gland. The cells from the sebaceous glands in which sebum originates will be the sebocytes. The combination of the keratin and the sebum produces a plugging of the mouth or opening with the follicular canal and papules are formed by inflammation across the comedones. Depending upon their education of inflammation, pustules, cysts, nodules, granulomatous reactions, scars and keloids may develop.

There is now a cream with a naturally occurring serum that works well for cystic acne on back and severe acne

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